Transient Cardiomyopathy in a Patient With Coronavirus Disease-2019.
Identifieur interne : 000035 ( Main/Exploration ); précédent : 000034; suivant : 000036Transient Cardiomyopathy in a Patient With Coronavirus Disease-2019.
Auteurs : B K Anupama [États-Unis] ; Simant S. Thapa [États-Unis] ; Ioana Amzuta [États-Unis]Source :
- Journal of investigative medicine high impact case reports [ 2324-7096 ]
Descripteurs français
- KwdFr :
- Betacoronavirus (MeSH), Cardiomyopathies (complications), Cardiomyopathies (immunologie), Cardiomyopathies (thérapie), Débit systolique (MeSH), Fonction ventriculaire gauche (MeSH), Humains (MeSH), Immunisation passive (méthodes), Infections à coronavirus (complications), Infections à coronavirus (immunologie), Infections à coronavirus (métabolisme), Infections à coronavirus (thérapie), Mâle (MeSH), Pandémies (MeSH), Pneumopathie virale (complications), Pneumopathie virale (immunologie), Pneumopathie virale (thérapie), Résultat thérapeutique (MeSH), Sujet âgé (MeSH).
- MESH :
- immunologie : Cardiomyopathies, Infections à coronavirus, Pneumopathie virale.
- métabolisme : Infections à coronavirus.
- méthodes : Immunisation passive.
- thérapie : Cardiomyopathies, Infections à coronavirus, Pneumopathie virale.
- complications : Betacoronavirus, Cardiomyopathies, Débit systolique, Fonction ventriculaire gauche, Humains, Infections à coronavirus, Mâle, Pandémies, Pneumopathie virale, Résultat thérapeutique, Sujet âgé.
English descriptors
- KwdEn :
- Aged (MeSH), Betacoronavirus (MeSH), Cardiomyopathies (complications), Cardiomyopathies (immunology), Cardiomyopathies (therapy), Coronavirus Infections (complications), Coronavirus Infections (immunology), Coronavirus Infections (metabolism), Coronavirus Infections (therapy), Humans (MeSH), Immunization, Passive (methods), Male (MeSH), Pandemics (MeSH), Pneumonia, Viral (complications), Pneumonia, Viral (immunology), Pneumonia, Viral (therapy), Stroke Volume (MeSH), Treatment Outcome (MeSH), Ventricular Function, Left (MeSH).
- MESH :
- complications : Cardiomyopathies, Coronavirus Infections, Pneumonia, Viral.
- immunology : Cardiomyopathies, Coronavirus Infections, Pneumonia, Viral.
- metabolism : Coronavirus Infections.
- methods : Immunization, Passive.
- therapy : Cardiomyopathies, Coronavirus Infections, Pneumonia, Viral.
- Aged, Betacoronavirus, Humans, Male, Pandemics, Stroke Volume, Treatment Outcome, Ventricular Function, Left.
Abstract
A 66-year-old male patient with coronavirus disease-19 (COVID-19) developed cardiogenic shock with echocardiographic evidence of decreased left ventricular ejection fraction and global hypokinesia concomitant with a robust systemic inflammatory response. Following the administration of convalescent plasma therapy and inotropic support, left ventricular function recovered fully in accordance with the decrease in the concentration of the inflammatory markers. Thus, we demonstrate the presence of transient reversible cardiomyopathy in a patient with severe COVID-19 and illustrate the association of acute cardiac dysfunction with profound systemic inflammation among COVID-19 patients.
DOI: 10.1177/2324709620947577
PubMed: 32762374
Affiliations:
Links toward previous steps (curation, corpus...)
Le document en format XML
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<term>Cardiomyopathies (immunology)</term>
<term>Cardiomyopathies (therapy)</term>
<term>Coronavirus Infections (complications)</term>
<term>Coronavirus Infections (immunology)</term>
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<term>Humans (MeSH)</term>
<term>Immunization, Passive (methods)</term>
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<term>Pandemics (MeSH)</term>
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<term>Pneumonia, Viral (immunology)</term>
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<term>Cardiomyopathies (thérapie)</term>
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<term>Fonction ventriculaire gauche (MeSH)</term>
<term>Humains (MeSH)</term>
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<term>Infections à coronavirus (complications)</term>
<term>Infections à coronavirus (immunologie)</term>
<term>Infections à coronavirus (métabolisme)</term>
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<front><div type="abstract" xml:lang="en">A 66-year-old male patient with coronavirus disease-19 (COVID-19) developed cardiogenic shock with echocardiographic evidence of decreased left ventricular ejection fraction and global hypokinesia concomitant with a robust systemic inflammatory response. Following the administration of convalescent plasma therapy and inotropic support, left ventricular function recovered fully in accordance with the decrease in the concentration of the inflammatory markers. Thus, we demonstrate the presence of transient reversible cardiomyopathy in a patient with severe COVID-19 and illustrate the association of acute cardiac dysfunction with profound systemic inflammation among COVID-19 patients.</div>
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<Abstract><AbstractText>A 66-year-old male patient with coronavirus disease-19 (COVID-19) developed cardiogenic shock with echocardiographic evidence of decreased left ventricular ejection fraction and global hypokinesia concomitant with a robust systemic inflammatory response. Following the administration of convalescent plasma therapy and inotropic support, left ventricular function recovered fully in accordance with the decrease in the concentration of the inflammatory markers. Thus, we demonstrate the presence of transient reversible cardiomyopathy in a patient with severe COVID-19 and illustrate the association of acute cardiac dysfunction with profound systemic inflammation among COVID-19 patients.</AbstractText>
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